Neuropathic Pain Induces Interleukin-1β Sensitive Bimodal Glycinergic Activity in the Central Amygdala
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MDPI AG
Citation
International Journal of Molecular Sciences, 23(13), 7356. https://doi.org/10.3390/ijms23137356
Abstract
Neuropathic pain reduces GABA and glycine receptor (GlyR)-mediated activity in spinal and supraspinal regions associated with pain processing. Interleukin-1β (IL-1β) alters Central Amygdala (CeA) exci...
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Except where otherwised noted, this item's license is described as Creative Commons Attribution 4.0 International
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